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This report will focus on the widening skills gap within the sector and the training needs of the public sector. It is being led by the EEF’s Skills and Employment Board, with the support of the new Public Service Innovations (PSI) project, which will deliver a major new national report and set of recommendations to improve skills and employment outcomes for the sector.
Note that in the UK, the government does not have a responsibility to provide public sector workers with education or training, but it can agree to pay for it through its education budget.
The report will also look at how best we can ensure that every public sector worker is trained and able to go on to work in the government sector.
The report is expected to be published in the summer of 2017.Rationale
The development of novel diagnostics for the detection of prion diseases is driven by human prion disorders, which are invariably fatal. Antibodies against PrP can be detected in prion diseases, but are non-specific and do not distinguish between ‘infectious’ and ‘uninfectious’ PrP. Despite the strong B-cell response against prion protein, it is still uncertain if the humoral response is protective and how exactly it impacts the disease.
Objectives
We investigate the systemic and cellular immune response to prion infection in experimentally infected mice by using microarrays and mass spectrometry to determine whether specific immune pathways are activated in response to prion infection.
Experimental design
C57BL/6 mice, Tg mice and knock-out mice were infected with scrapie prions after different administration routes. At 21 dpi, immune profiles of spleens and lymph nodes were analyzed by microarrays, which revealed that naïve PrP transgenic mice and naïve wild-type mice exhibited completely different gene expression profiles in response to prion infection.
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